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Overcoming Bias Commenter's avatar

Stories are powerful, but can we attribute their power to genes or to experience?

this is an ill-formed question. on some level, the power of stories is obviously genetic. try telling a mouse a story, and see how much he gets out of it. the reason the mouse doesn't get it is because the basic machinery (genetically encoded) just isn't there. if human brains respond to stories, it's because our genetically encoded machinery is capable of it.

Our brains are adapted to a universe where time is sequential and people act with motives. Maybe your question is: if we were transplanted to a universe where that were not the case, would we still respond to stories better than exposition? When you put it like that, it's maybe an interesting rhetorical question (my answer would be yes, of course), but there's no way of testing it.

It may be the case that there is individual variation in the degree to which people respond to stories vs. exposition (you could imgaine traits like patience and intelligence playing a role). In this case, it is theoretically possible to identify the individual genes themselves that play a role in the bias.

I just thought it unlikely that commonplace random mutation from crossover and the like would be so uniform ("every single fetus") as to wipe the pinky toe out of the gene pool. In other words, without some selective pressure (altering reproduction rates), I don't see the "random mutation" story accounting for a loss or reduction of pinky toes, especially in the short term.

you mean random mutation (crossing-over is something different). in the short term, you're right, the pinky toe isn't going anywhere. But if there's no selective pressure maintaining the pinky toe (that is, if it is "neutral"), there no reason why it couldn't disappear in the long run (thousands/ hundreds of thousands of years). Of course, it could still be around-- the evolutionary process is stochastic.

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Overcoming Bias Commenter's avatar

Pete,

Your explanation was clearer, though my (2) was trying to make the same point about the "increase of entropy in the 'pinky toe genes.'" I just thought it unlikely that commonplace random mutation from crossover and the like would be so uniform ("every single fetus") as to wipe the pinky toe out of the gene pool. In other words, without some selective pressure (altering reproduction rates), I don't see the "random mutation" story accounting for a loss or reduction of pinky toes, especially in the short term.

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